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UPI : reported that Study shows Alzheimer’s disease has roots in the womb

As it stated in Jan. 27 (UPI) — Research from the University of British Columbia has found that biochemical reactions in brain tissue that cause Alzheimer’s disease can start in the womb.
A mild vitamin A deficiency increased the production of amyloid beta, a protein that forms plaques that kill neurons in Alzheimer’s disease.
“In some cases, providing supplements to the newborn Alzheimer’s disease model mice could reduce the amyloid beta level and improve learning and memory deficits,” Song said.
Conversely, vitamin A supplements given to newborns could slow the progression of the disease.
The study found that a vitamin A deficiency in the womb caused mice to perform worse on standard tests of learning and memory.

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Roots of Alzheimer’s disease can extend as far back as the womb: Vitamin A deficiency could ‘program’ brain tissue — ScienceDaily

Roots of Alzheimer's disease can extend as far back as the womb: Vitamin A deficiency could 'program' brain tissue -- ScienceDaily

The researchers found that even a mild vitamin A deficiency increased the production of amyloid beta, the protein that forms plaques that smother and ultimately kill neurons in Alzheimer’s disease.
“In some cases, providing supplements to the newborn Alzheimer’s disease model mice could reduce the amyloid beta level and improve learning and memory deficits,” said Song.
These new findings, based on studies of genetically-engineered mice, also demonstrate that supplements given to newborns with low levels of vitamin A could be effective in slowing the degenerative brain disease.
He also found that these mice, when deprived of vitamin A, performed worse as adults on a standard test of learning and memory.
Biochemical reactions that cause Alzheimer’s disease could begin in the womb or just after birth if the fetus or newborn does not get enough vitamin A, according to new research from the University of British Columbia.

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Vitamin A deficiency linked to Alzheimer’s disease

Mouse studyIn the mouse model of Alzheimer’s disease, mice were fed a diet marginally deficient in vitamin A, as this was more common in humans than complete vitamin A deficiency.
This study in both humans and mice aimed to see whether vitamin A deficiency may have a role in the development of Alzheimer’s disease.
The findings are of interest and worthy of further study, but provide no evidence that vitamin A deficiency causes Alzheimer’s.
They found feeding mice vitamin A-deficient diets increased the development of abnormal clumps of protein associated with the condition.
New research involving both mice and humans looked at the link between vitamin A deficiency, brain development and Alzheimer’s risk.

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