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There’s a close relationship between sleep and protection from the risk of heart attacks

Surprising Sleep Discoveries]”If you have several heart disease risk factors, taking care of your sleep and consulting with a clinician if you have insufficient sleep is important if you want to lower your risk of death from heart disease or stroke,” lead study author Julio Fernandez-Mendoza, an assistant professor of psychiatry at Penn State College of Medicine, said in a statement.
[Where Is Heart Disease Risk the Highest and Lowest?
For people who are at risk for heart disease and diabetes, getting enough sleep may be especially important: Snoozing for less than 6 hours per night may increase their risk of premature death, a new study finds.
At the lab, the researchers also looked at whether the participants had metabolic syndrome.
The researchers looked at the relationship between metabolic syndrome, how long the participants slept at the study’s start and their risk of death during the 17 years.

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4 Surprising Dietary Links To Death From Heart Disease

4 Surprising Dietary Links To Death From Heart Disease

Major Dietary Factors for Death from Heart Disease A low intake of nuts and seeds was the leading dietary risk factor for death from cardiovascular disease (CVD), accounting for 11.6 percent of deaths due to CVD.
But, perhaps the most interesting findings were the breakdown of how foods or lack of certain foods contributed to death from heart disease.
They found that nearly half of all deaths from heart disease were the result of diet and easily preventable from dietary changes alone.
And, studies show that a healthy diet can reduce the risk of dying from cardiovascular disease by nearly half.
Even today it astounds me that so many people with heart disease are encouraged by their doctors and other health professionals to eat a diet that is simply not founded in research.

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Targeting ANGPTL3 Could Lead to Development of Heart Disease Therapies

NEW YORK (GenomeWeb) – Researchers from two pharmaceutical companies are pursuing drugs that target the ANGPTL3 gene in a bid to treat heart disease.
The companies hope to recapitulate, to some degree, the lower triglyceride and cholesterol levels seen among people with naturally occurring loss-of-function mutations in ANGPTL3.
People who are homozygous for such mutations have some 70 percent lower plasma triglycerides and LDL cholesterol levels than those without.

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